That is interesting because caffeine as low as ~80mg is one of my triggers for AFIB and arrhythmia and absolutely wrecks me within 15 minutes of consumption. I was not a regular coffee drinker though, so maybe the "regular drinkers" bit in that statement is the significant factor here.
Under "Conclusions and Research Needs":
> Last, in terms of applications of the potential beneficial effects of coffee intake, it is worth studying whether introducing or initiating regular coffee consumption among current abstainers yields positive cardiovascular health benefits.
In other words, more study is needed to determine whether abstainers should start consuming caffeine.
Additionally, the study suggests much of the cardiovascular benefits are from non-caffeine compounds in coffee/tea:
> Although much of the evidence on caffeine derives from studies of coffee consumption, coffee contains numerous bioactive compounds beyond caffeine, including chlorogenic acids, diterpenes, trigonelline, and melanoidins. In large cohort studies, inverse associations between coffee intake and risk of type 2 diabetes and cardiovascular disease have also been observed for decaffeinated coffee, supporting a role for noncaffeine components.
However, some arrhythmologists and electrophysiologists sing a different tune. When I was diagnosed with something complex and novel that results in ventricular arrhythmia (PVCs and VT), my EP was, to put it mildly, uninterested in my consumption of caffeine. He said that, at worst, it made no significant difference. That was a few years ago. I was pleasantly surprised.